J series (2006) 11 episodes

Official Website: http://wwwz.fujitv.co.jp/iryu/index2.html

Cast:
Kenji Sakaguchi - Asada Ryutaro
Inamori Izumi - Katou Aki
Koike Teppei - Ijuuin Noboru
Kitamura Kazuki - Kirishima Gunji
Abe Sadayo - Arasa Monji
Mizukawa Asami - Satohara Miki

Story:
adaptasi dari manga dengan judul yg sama. Cerita soal dokter Asada, surgoen, sangat berbakat tapi tidak mau ikutin sistem yg sudah ada (emg sistem yg korup). Asada di rekrut sama dr. Aki untuk membentuk team operasi untuk operasi jantung dengan metode Batista. Aki ingin jadi professor, karena itu perlu bantuan Asada dalam melakukan thesisnya. Asada sulit dapat perkerjaan di Jepang sekembali dari NGO karena attitudenya.

http://www.sakaguchikenji.com/ - Official Website nya Kenji Sakaguchi.

THE BATISTA OPERATION

The newly developed Batista operation (named after Brazilian surgeon Dr. Randas Batista, who introduced the procedure in 1995) is a left ventricular remodeling operation for patients with end-stage heart failure. In this innovative operation which is technically called partial left ventriculectomy, or reduction left ventriculoplasty, a portion of heart muscle is removed and the geometry of the heart remodeled, thus improving the heart's ability to pump blood.

When the heart starts to fail, it increases in volume, occasionally to twice its original size. As a result, it loses its ability to pump blood effectively. Why this happens is not known. Typically, the only cure for patients with end-stage heart failure is a heart transplant. In the United States, these patients have about a one in ten chance of receiving a donor heart.

Now the Batista operation may provide these patients with relief of heart failure, and prolong their lives until a heart transplant can be performed. The early experience with this new operation is encouraging. Not all patients with end-stage heart failure can be candidates for it, but there are certain patients who might particularly benefit from it, especially those with very dilated (enlarged) hearts.

As of September 1998, Dr. Irvin B. Krukenkamp, professor of surgery and chief of cardiothoracic surgery, has successfully performed six Batista operations. With no mortalities, his results compare favorably to what has been achieved at other heart centers nationwide.

The Procedure Itself

The Batista operation is an open-heart operation performed with the aid of a heart-lung machine to maintain circulation while the heart is stopped. The essence of the procedure is to remove a wedge of left ventricle muscle (weighing about 40 to as much as 250 grams, or 1.5 to 9 ounces) and stitch together the two edges of the ventricle. In this manner the size of the left ventricle is reduced, and the ventricle is remodelled as well.

In addition, sometimes the mitral valve (which lies between the left ventricle and left atrium, and often leaks in end-stage heart failure) may need to be either repaired or removed and replaced with an artificial valve. In two of Dr. Krukenkamp's patients, mitral valve repair was done, rendering the valves completely competent.

In patients who have heart rhythm disturbances as well, an artificial implantable defibrillator device may be implanted. If the coronary arteries are diseased, they may be grafted, too (CABG operation).

In left ventricular failure, the muscle cells in the wall of the left ventricle have been stretched beyond physiologic limits. The Batista operation, by removing a wedge of the ventricular wall, reduces the circumference of the ventricle, and the size of the ventricle cavity. It changes the relationships of pressure and stress on the wall.

Consequently, the individual muscle fibers are restored to their normal length, and now come within the normal physiologic limit. They thus function more effectively in pumping blood, and in this way, heart failure is relieved.

In such a paradoxical manner, removal of heart muscle tissue actually helps the heart pump more effectively. The operation, as described above, is a radical departure from conventional thinking. Removal of too much muscle can weaken the heart and hasten death. It perhaps has the most potential benefit for patients with terminal heart failure caused by a disease of heart muscle (cardiomyopathy) and with a dilated left ventricle, who are not adequately helped by medication and are awaiting a heart transplant.

Bridge to Transplantation

The Batista operation is not an alternative to heart transplantation. It certainly helps most patients for some time. In a potential heart transplant candidate, the Batista operation buys some time to allow a more suitable and better matched donor heart to be found. Thus, it may serve as a useful bridge to transplantation.

Today, the only other alternative bridges to transplantation are the use of a ventricular assist device (VAD) and another new operation called dynamic cardiomyoplasty, in which muscle taken from the patient's back is wrapped around the heart and synchronized with a special pacemaker (cardiomyostimulator) to make it function like heart muscle.

VAD implantation is an extremely expensive option, and has a lot of complications and morbidity. Dynamic cardiomyoplasty is currently being studied in clinical trials, and its efficacy has not yet been established; possible side effects of this operation include body rejection phenomena/local tissue reaction, muscle and nerve stimulation, infection, erosion of the cardiomyostimulator/leads through the skin, transvenous lead- related thrombosis, embolism, and cardiac tamponade.

The Batista operation may therefore be a good option for treating end-stage heart failure. Ultimate heart transplantation, however, still remains an eventuality to be faced.

The future role of the Batista operation is not yet known. Scientific analysis of data from patients who have undergone this procedure is still in a very early stage. Without long-term follow-up and extensive analyses of different factors, the ultimate place of the Batista procedure in the management of heart failure cannot be determined. But with the extensive use of this new option for different stages and types of heart failure, such data will soon be available.

source: http://www.uhmc.sunysb.edu/surgery/batista1a.html

more info:

Pioneers of Heart Surgery
For most of history, the human heart has been regarded as a forbidden organ too delicate to tamper with. It might have remained so, were it not for World War II. Military doctors, facing injury and suffering on a massive scale, pioneered advances in antibiotics, anesthesia and blood transfusions -- advances that would usher in the age of modern surgery.

One of the first surgeons to use these improved techniques to gain access to the heart was Dr. Dwight Harken, a young U.S. Army surgeon. Many of Harken's patients were young soldiers evacuated from the European front with shell fragments and bullets lodged inside their hearts. To leave the shrapnel in was dangerous, but removing it was almost surely fatal. Harken began operating on animals, trying to develop a technique that would allow him to cut into the wall of a still beating heart, insert a finger, locate the shrapnel and remove it. All of his first 14 animals subjects died. Of the second group of 14, half died. Of the third group of 14, only 2 died. Harken felt ready to try the technique on humans. All of his patients survived, proving that the human heart could be operated upon.

It wasn't long before surgeons began wondering if Harken's technique might be applied to defective heart valves. In 1948, within days of each other, Harken and a Philadelphia surgeon, Dr. Charles Bailey, independently reported on a daring procedure to correct mitral stenosis: a condition where the mitral valve (see Hot Science) is narrowed and won't open properly. Just as with the soldiers, a small hole was cut in the side of a beating heart and a finger was inserted to find and very carefully widen the narrowed valve. Early results were disastrous, with the majority of patients dying. Gradually, though, surgeons improved their technique and the procedure became quite safe. This kind of blind surgery -- or closed heart surgery -- spread to hospitals around the world.

Impressive as the technique was, it made little difference to patients suffering from more serious heart defects -- children born with congenital heart disorders, breathless and blue and condemned to an early death -- or victims of rheumatic fever whose heart valves were narrowed or stuck. If surgeons couldn't work on the heart from the inside, nothing could be done. But how could surgeons open up the heart without their patients bleeding to death? Temporarily stopping a patient's circulation only gave doctors about four minutes to work before brain damage from oxygen deprivation took place.

At the University of Minnesota, a young Canadian surgeon named Dr. Bill Bigelow came up with the first workable, if highly bizarre, answer. He had noticed how hibernating animals, like ground hogs, survived the bitterly cold Canadian winters. Their hearts beat slower, allowing them to survive for months without food. Wondering if cold might be the key to operating inside the heart, Bigelow began animal experiments and found that when dogs were cooled, open heart surgery could be done for long periods -- much longer than four minutes -- and they didn't die. He showed that at lower temperatures, the tissues of the body and brain didn't need as much oxygen, and could survive without oxygenated blood for longer.

On September 2, 1952, two University of Minnesota surgeons, Dr. Walton Lillehei and Dr. John Lewis, attempted the first open heart surgery on a five-year-old girl who had been born with a hole in her heart. Anaesthetized to stop her shivering, the girl was cooled by a special blanket until her body temperature reached 81 degrees F. At this temperature, she could survive without a pumping heart for ten minutes, not four. Clamping the inflow to her heart so that it emptied of blood, Lillehei and Lewis cut open her heart, which was still slowly beating, and quickly sewed up the hole. With the repaired heart working properly for the first time in her life, the girl was then immersed in a bath of warm water to bring her body temperature back to normal. The operation was a success.

The "hypothermic approach" became very successful in treating small heart defects. But all too often, surgeons opened hearts to find more complex defects -- defects that couldn't be repaired in 10 minutes. With the clock ticking away, they did what they could, but it was clear that a better approach needed to be found.

The dream of building a machine to take over the function of the heart and lungs during surgery had existed before World War II. Early prototypes, built by pioneers like Dr. John Gibbon in Great Britain, were cumbersome and dangerous -- often leaking blood, damaging blood cells and causing air embolisms. It wasn't until 1958, when a system that involved bubbling blood was perfected, that "heart-lung" machines came of age. Dr. Dennis Melrose of London further increased chances for success when he pioneered an injection that stopped the heart from beating during surgery.

Now surgeons had time to work on a heart that was not only empty of blood, but which wasn't moving. And they had time to correct the most serious abnormalities. Holes which were too big to be sewn up were patched. Where valves were damaged beyond repair, artificial valves were put in. Blocked arteries were bypassed. Weakened arteries were replaced altogether. Modern heart surgery seemed unstoppable.

But a major problem still remained -- what to do for patients whose very heart muscle was diseased beyond repair? Could these patients be given new hearts? By 1966, heart surgeons were ready to take on the challenge. Most, like Dr. Michael De Bakey of Houston, thought the answer lay in artificial hearts. But the future would lie in a different direction: heart transplants. Kidneys had been transplanted successfully as early as 1963, after the complexities of tissue rejection were solved with drugs that suppressed the immune response. If the barrier had been breached for the kidney, why not for the heart?

In December of 1967, a South African surgeon, Dr. Christiaan Barnard, transplanted the heart of a 23-year-old woman killed in a motor vehicle accident into the chest of a middle-aged man. He lived for eighteen days, until the powerful drugs used to suppress rejection weakened him and he died of pneumonia. The second patient to receive a heart transplant, at the hands of Dr. Adrian Kantrowitz in the United States, lived only six hours. But Dr. Barnard's next heart-transplant patient lived for 18 months and became a symbol of hope for victims of heart disease. All over the world patients were asking and receiving the new miracle operation.

But these surgical triumphs proved short-lived. Patients began dying of either rejection or infection. By 1971, 146 of the first 170 heart transplant recipients were dead. What first looked like another surgical miracle had turned into a disaster. Heart surgeons who had promoted the operation admitted defeat.

Only one American surgeon would continue -- Dr. Norman Shumway. Throughout the 1970's, he built a team of scientists and doctors to tackle the complex biological problem of tissue rejection in a careful, scientific manner. His team devised a way of spotting rejection attacks, by feeding a catheter into the heart and removing a piece of heart muscle for examination. Only when signs of rejection were seen were doses of the dangerous immuno-suppressive drugs increased. And Shumway benefited from a chance discovery made in another part of the world.

In the soil of Norway's Hardaanger fjord, a fungus was found which contained a compound that would revolutionize transplant surgery. The substance, called cyclosporin, appeared to have exquisite immuno-suppressant properties -- controlling organ rejection without knocking out all resistance to infection. In the hands of Dr. Shumway, cyclosporin transformed the picture for heart transplant recipients. Hospitals around the world began to re-open their heart transplant units and their patients began to survive and prosper.

But this breakthrough has come with limitations, too. The problem with heart transplants now has become finding enough hearts. Today in the United States alone, 2 million people suffer from congestive heart failure. When drug treatments fail, transplants are the best hope. But less than 2,500 donor hearts are available each year, leaving thousands of patients desperate for an alternative.

In 1994, Dr. Randas Batista of Brazil devised a radical new surgical technique to treat a common form of heart failure for people with enlarged hearts. Normally, oxygen-rich blood flows into the left side of the heart from the lungs (see Hot Science). The left ventricle is responsible for pumping the blood out to the rest of the body. When the heart becomes diseased, it sometimes dilates or swells. The contractions become sluggish and the left ventricle is unable to squeeze out enough blood. Blood backs up in the heart and the lungs, resulting in congestive heart failure.

Batista's idea was to cut a swath out of the left ventricle and sew the chamber back together, thereby reducing its size and increasing its efficiency. Gradually, news of Batista's radical approach spread and, currently, a small number of surgeons around the world are experimenting with the procedure. Their results, so far, have been mixed. More time and innovation are needed before it's known whether this technique will be the next milestone in the history of heart surgery.

Source: http://www.pbs.org/wgbh/nova/heart/pioneers.html (ada gambar2nya disini. interesting)

wahhh...panjang juga nih bacanya...musti print ahhhh...baca di rumah aja besok baru bahas

btw, untuk doramanya just two words, kakkoi...sugoi... keren bangetttttt...apalagi operasinya

yoi panjang itu batista, man man kan la....
back to dorama, keren ya doramanya....
asada sensei sugoi desu ne...

pengen nonton lagi yang kayak gini...

udah kelar bacanya...
interesting, sambil baca itu ttg operasi terbayang si asada lagi operasi jantung...
nonton dorama begini nambah ilmu, gue suka...

udah kelar baca nihhh...wahhh...panjang juga ceritanya dan ternyata jantung itu rumit juga yah itu artikel di atas menarik, mayan deh nambah ilmu hehehe...

udah kelar nonton juga...wahhhh kakkoi dan sugoi deh...operasi yang terakhir kerennnnn...

salut juga lhooo...ternyata mereka bisa diam2 merencanakan operasi dadakan disaat para atasan sedang meeting salut dah sama Asada-kun itu Ijuuin-kun juga pas dilihat dia kerjasama sama si Kirishima wahhhh...udah ketebak sih dan untungnya benar tebakannya

btw, tadinya sempat mikir si Teppei-kun penampilan seperti itu mirip sapa yahhh...sampe akhirnya nyerah ternyata pas waktu si Kihara manggil dia Harry Potter baru deh ngeh kalo dia mirip si Daniel yang peranin si HP dilihat2 lagi emang mirip oiii...

gile wi, ngebut neh semalem...
itu rada spoiler ya yg di atas

tapi memang operasi terakhir si asada memang hebat banget...
gue kemaren ke rental liat2 komik team medical dragon, masih mempertimbangkan mo sewa or enggak gitu...
abis asada di film lebih cakep daripada di komik

iya, kemarin jam 01.00 tuh makanya sekarang ngantuk bangetttttt...tapi kerjaan mayan nih hari jadi gak terpengaruh banget, kalo gak ada kerjaan bisa tidur kali cuman kayaknya mulai dari eps 8 tuh teks-nya rada2 yah tapi yang penting ngerti dan dari kemarin gak bisa berhenti ngomong sugoi kakkoi nih

yang spoiler udah me edit tuh

btw, ayo donkkk komiknya ada yang beli gitu ntar me pinjam

baru selesai td, hrsnya kemaren sih tp di awal2 eps terakhir ketiduran hehe lumayan ceritanya , Asada keren oii.. Tp serunya pas lg operasi doang, konflik batinnya Asada sendiri kurang disorot

semalam selesai nonton ulang and the magic has gone ... mungkin hrs tunggu 3-4 bulan lagi baru feel the same magic.. ha,ha,ha,ha... tp emg bener si Asada keren banget, cocok karakter dia sama pemainnya.

baru mulai liat...setelah dipinjemin sama Dewi..
G paling suka series ttg RS, asal bukan bikinan Indo ( emang ada ? ) operasi2nya realistis, niat gitu bikinnya.
Asada-nya keren, apalagi kalo lg ga cukur..
tp kenapa kalo latihan operasi di pikiran yg di atap itu mesti topless gitu seh? ga masuk angin apa? hehehe
BTW, g malah suka tampangnya Kirishima sensei.. suka cowok bertampang jahat.. ( not to mention also like Prada style-guy... )

baru liat profile-nya Kenji di website-nya, tinggi 185..impressive..tahun 75? :0 hehehe..
T/185cm　B/93cm　W/77cm　H/92cm　S/29cm --->> apa artinya sih? t=tinggi? b? w? n S? 29cm??? apanya tuh?????

set dah djes, sampe dicari segitunya...
heheh Kenji itu aniki ya... sip sip belakangan semuanya ototo semua...

ga dicari, kan tina ada kasih linknya di 1st page? jadi daku cuman buka aja..trus baca2 de...aniki=cakep ya? iyaa...

aniki = kakak
hehe usia lebih tua dari gue, cocok di HM-in

oiya, aniki kakak, kan kemaren abis liat di fugoh..
mesti belajar japanese lg nih...jadi malu

trus ototo apaan?
-- tadinya ga mau malu jadinya diem2 aja.. eh ga dibahas2..

ototo = adik cowok (hehe kalo gak salah lho, ini berdasarkan ingatan gue yg parah )

benul! otoko = lelaki. otouto = adik lelaki.. nisan/aniki = kakak lelaki. neechan = kakak perempuan, imouto = adik perempuan.. he,he,he.. anata = dear... banyak2 lah jdorama dan anime

aniki itu kan biasanya untuk kakak laki2 pertama, kalo yang kedua dstnya biasanya nisan